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const regex = /\d+\s?(women|men)/gm; // Alternative syntax using RegExp constructor // const regex = new RegExp('\\d+\\s?(women|men)', 'gm') const str = `OR I GI N AL AR TI CL E Home-based interval training increases endurance capacity in adults with complex congenital heart disease Camilla Sandberg RPT, PhD 1,2 | Magnus Hedstr€ om MD 1 | Karin Wadell RPT, PhD 2 | Mikael Dellborg MD, PhD 3 | Anders Ahnfelt MD 3 | Anna-Klara Zetterstr€ om RPT 4 | Amanda € Ohrn RPT 4 | Bengt Johansson MD, PhD 1 1 Heart Center and Department of Public Health and Clinical Medicine, Umeå University, Umeå, Sweden 2 Department of Community Medicine and Rehabilitation, Physiotherapy, Umeå University, Umeå, Sweden 3 Department of Molecular and Clinical Medicine, Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden 4 Department of Physiotherapy and Occupational Therapy, Sahlgrenska University Hospital, Gothenburg, Sweden Correspondence Camilla Sandberg, RPT, PhD, Heart Center and Department of Public Health and Clinical Medicine, Umeå University, SE-90185, Umeå, Sweden. Email: camilla.sandberg@umu.se Funding information Swedish Heart-Lung Foundation, Grant/ Award Numbers: 20100355, 20130472; Heart Foundation of Northern Sweden; Research Foundation of The Swedish Heart and Lung Association, Grant/Award Num- ber: E116/12, E115/13, E129/14; Research Foundation of Healthcare Professions within Cardiology, Umeå University; Väster- bottens läns landsting, Umeå, Sweden, Grant/Award Number: 316351; and ALF- LUA grants at Sahlgrenska University Hos- pital, G€ oteborg, Sweden Abstract Objective: The beneficial effects of exercise training in acquired heart failure and coronary artery disease are well known and have been implemented in current treatment guidelines. Knowledge on appropriate exercise training regimes for adults with congenital heart disease is limited, thus further studies are needed. The aim of this study was to examine the effect of home-based interval exercise training on maximal endurance capacity and peak exercise capacity. Design: Randomized controlled trial. Methods: Twenty-six adults with complex congenital heart disease were recruited from special- ized units for adult congenital heart disease. Patients were randomized to either an intervention group—12 weeks of home-based interval exercise training on a cycle ergometer (n516), or a con- trol group (n510). The latter was instructed to maintain their habitual physical activities. An incremental cardiopulmonary exercise test and a constant work rate cardiopulmonary exercise test at 75% of peak workload were performed preintervention and postintervention. Results: Twenty-three patients completed the protocol and were followed (intervention n513, control n510). Postintervention exercise time at constant work rate cardiopulmonary exercise test increased in the intervention group compared to controls (median[range] 12[–4 to 52]min vs 0 [–4 to 5]min, P5.001). At incremental cardiopulmonary exercise test, peak VO 2 increased 15% within the intervention group (P5.019) compared to 2% within the control group (P5.8). How- ever, in comparison between the groups no difference was found (285[–200 to 535] ml/min vs 17 [–380 to 306] ml/min, P5.10). In addition, peak workload at incremental cardiopulmonary exer- cise test increased in the intervention group compared to controls (20[–10 to 70]W vs 0[–20 to 15]W, P5.003). Conclusion: Home-based interval exercise training increased endurance capacity and peak exer- cise capacity in adults with complex congenital heart disease. Aerobic endurance might be more relevant than peak oxygen uptake with regard to daily activities, and therefore a more clinically rel- evant measure to evaluate. K EY WO RD S adult, cardiopulmonary exercise testing, congenital heart disease, constant work rate, exercise training, interval training 254 | V C 2017 Wiley Periodicals, Inc. wileyonlinelibrary.com/journal/chd Congenital Heart Disease. 2018;13:254–262. Received: 3 January 2017 | Revised: 21 August 2017 | Accepted: 19 October 2017 DOI: 10.1111/chd.12562 1 | INTRODUCTION During the past decades there have been substantial improvements in survival and reduction in the need of reoperations for adults with congenital heart disease (CHD), especially among those with more complex lesions. 1–3 There is a well-known impairment in exercise capacity in these patients compared to healthy controls. 4,5 To what extent this impairment is due to abnormal cardiovascular physiology, per se, or to other factors such as physical inactivity or lack of exercise training, that is, deconditioning, is unknown. Fur- thermore, reduced exercise capacity is an important prognostic predictor of cardiovascular morbidity and mortality. 6 In addition, impaired muscle function has been observed, especially in adults with CHD that have complex lesions. 7–10 According to current rec- ommendations of the European Society of Cardiology, adults with CHD are encouraged to be physically active and exercise training should be prescribed individually. 11 However, due largely to the low numbers of studies on effects of exercise training in adults with CHD, there are no specific recommendations regarding train- ing mode, intensity, duration, and frequency. 12,13 Previous studies show that exercise training seems to be safe and to have positive effects on aerobic exercise capacity without adverse effect on ven- tricular function. 14–21 Home-based continuous moderate exercise training on an cycle ergometer increased the exercise capacity in adults with a systemic right ventricle. 17 In patients with acquired heart failure, interval training was shown to improve exercise capacity more than continuous moderate exercise training. 22 Inter- val training at moderate to high intensity (75%–95% of maximum HR) was also reported to be safe and increase exercise capacity in these patients. 15,18 To improve adherence to study protocol, home-based exercise training has been used in a number of previ- ous studies. 17,23,24 Incremental cardiopulmonary exercise testing (incremental CPET) on a cycle ergometer or treadmill is frequently used and often considered the “gold standard” when evaluating aerobic exercise capacity and change in aerobic capacity after intervention. 25,26 Changes in peak aerobic capacity, total exercise time and peak work load are variables commonly used to report study results. 12 Constant work rate CPET is a method commonly used to evaluate endurance exercise capacity in patients with chronic obstructive pulmonary dis- ease (COPD) 27 and it has emerged as the most responsive test method to evaluate change in exercise capacity in these patients after rehabilitation intervention. 28 At present, this test has not been used to evaluate outcome of exercise training in the setting of adult CHD. The present study was a randomized controlled trial that used CPET aimed to evaluate the effect of home-based interval exercise training on submaximal exercise capacity (endurance) and peak exercise capacity (peak work load, peak oxygen uptake) in adults with complex CHD. The hypothesis was that interval training increases endurance capacity as well as peak exercise capacity in an intervention group compared to a control group. 2 | METHODS 2.1 | Study population Twenty-six patients (13 women) with complex CHD and reduced exer- cise capacity were recruited from specialized units for adults with CHD in the northern health care region (Umeå) and the region of Västra G€ otaland (G€ oteborg) in Sweden. The inclusion criteria were complex CHD, as defined by Erikssen et al., 1 (eg, palliated with variants of Fontan procedure [Fontan/TCPC], pulmonary atresia [PA], tetralogy of Fallot [ToF], congenitally corrected transposition of the great arteries [ccTGA], dextro-transposition of the great arteries repaired with Mustard or Sen- ning procedure [d-TGA]), clinically stable condition over the past 3 months, adult age (?18 years of age), and informed consent. The patients with PA, as well as those with ToF, had undergone surgical repair and were not cyanotic. The exclusion criteria were present strat- egy for executing exercise training?2 times/week aimed at increasing or sustaining exercise capacity, arrhythmia or other adverse events (eg, important symptoms, drop in blood pressure) at CPET, clinically relevant arrhythmia, intellectual disability or mental illness affecting independent decision making, extracardiac disease affecting physical activity, peak VO 2 >30ml/kg/min at run-in CPET, or no internet access. At the time of screening (October 25, 2012) the outpatient register of CHD in the northern health care region, 391 patients were registered. Seventy- seven patients met the inclusion criteria of complex CHD. Of these, 39 patients met at least one exclusion criteria. Thus, 38 patients were eligi- ble and were asked to participate; 17 declined participation, 1 was excluded due to arrhythmia (short periods of atrial fibrillation) and 1 was excluded due to peak VO 2 ?30 ml/kg/min at run-in CPET and 19 (53%) were finally included. There were no differences regarding age and sex between those who declined participation and those who par- ticipated. In the region of Västra G€ otaland, a convenience sample was collected, that is, when patients fulfilling the inclusion criteria were scheduled for a regular follow-up visit they were asked to participate. Thus, these patients were not strictly consecutively recruited. Eight patients were tested with run-in CPET, of these 1 was excluded due to arrhythmia. This gave an additional 7 patients. Finally, a total of 26 patients were included and were randomized to an exercise-training group, that is, intervention group (n516), or a control group (n510). Two patients that had been randomized to the intervention group dis- continued study participation due to personal reasons. For another patient, the exercise training was discontinued after 14 training sessions due to the patient experiencing discomfort and possible arrhythmia. Therefore, 23 patients consisting of 13 in the intervention group and 10 in the control group were followed after intervention. All patients gave their written informed consent for participation. The study was approved by the regional Ethics Review Board in Umeå (Dnr 2011-51- 31 M, 2011-03-29, and 2012-143-32 M, 2012-04-04). 2.2 | Exercise capacity At the incremental CPET peak VO 2 , peak work load, respiratory exchange ratio (RER) and peak heart rate were registered. 25 To ensure standardization, the participants were instructed that when rating ?17 SANDBERG ET AL . | 255 (very hard) on the Borg Rated Perceived Exertion scale, 29 this corre- sponded to perceived exertion of “not coping with an additional increase of work load.” The subsequent endurance test, constant work rate CPET, was performed at 75% of peak work load that had been achieved at the initial incremental CPET. The exercise test duration was the main outcome. 27,30 In one patient in the intervention group, the postintervention constant work rate CPET was not performed due to technical issues. Therefore, for this patient we only used the incre- mental CPET data. For pre- and postexercise tests the Jaeger Oxycon Pro CareFusion (GmbH, Hoechberg, Germany) or Schiller CS-200 Ergo- spirometry (Schiller AG, Baar, Switzerland) was used for analysis of breathing-gases. 2.3 | Randomization process The present study was a two-armed randomized controlled trial. Patients were randomly assigned to intervention or control group in the ratio of 2:1 that was applied by computer generated block random- ization. The group allocation sequence was kept in an opaque, sealed, and stapled envelope to prevent prior knowledge, and was revealed to patients and researchers after completion of run-in tests. At the first center, the technicians performing the tests and the physician (MH) analyzing the tests were blinded for group allocation. Furthermore, the patients were instructed not to reveal their group allocation. At the second center, the investigators were not blinded. However, the tests strictly followed the prespecified protocol. 2.4 | Exercise training protocol The exercise training was home-based and was performed three times a week for 12 weeks on a cycle ergometer with a manually adjusted brake system (Tunturi T 20/Tunturi, Tunturi- Hellberg Oy Ltd, Åbo, Fin- land or Bremshey BF3, Escalade Int. Ltd, Nottingham, UK). The time between completion of the run-in tests, and start of intervention was approximately 1 week due to delivery of the cycle ergometers. To indi- vidually adjust the intensity of the exercise training protocol, the train- ing heart rate (THR) was calculated according to the Karvonen method based on the individual peak heart rate. 31 In addition to the heart rate intervals, patients were instructed to achieve perceived exertion corre- sponding to BORG 15–16. 29 All patients randomized to exercise train- ing received one occasion with familiarization training. The exercise training had an initial 8 min warm-up without load or with very low load. During the first 2 weeks the protocol consisted of three intervals at THR 75%-80%, and thereafter four intervals. The duration of the inter- vals was also individually adjusted according to total exercise time dur- ing the initial constant work rate CPET. When the total time at constant work rate CPET was less than 5 minutes, the interval time was calculated as total exercise time minus 1 minute. The maximum interval time was 5 minutes. The intervals were separated by an active recovery periods of 3 minutes without load or with very low load (Sup- porting Information Figure S1). During the exercise training sessions, participants wore a heart rate monitor (Polar RS 300X, Polar Electro Oy, Kempele, Finland). The registered heart rate was regularly transferred to a personal webpage that was accessed by the physio- therapist and participant. A weekly contact by phone was used to pro- mote compliance, to provide feedback, and, when appropriate, to increase training time if a shorter interval time than 5 minutes. The par- ticipants were instructed to pay attention to symptoms such as dizzi- ness, palpitations, chest pain, and other experiences of discomfort, and if these occurred, abort exercise and contact investigators. The maxi- mum number of training sessions was 36 and the goal was that every participant should complete a minimum of 28 (78%) sessions. The patients randomized to control group were instructed to continue with their habitual physical activities. 2.5 | Questionnaires In addition to exercise test data, self-reported quality of life was eval- uated using the EuroQol Vertical Visual Analogue Scale (EQ-VAS). 32 The Hospital Anxiety and Depression scale (HADS) 33,34 was used for assessing prevalence of anxiety and depression. Finally, self-efficacy for exercise was evaluated using the Exercise Self-Efficacy Scale (ESE). 35 All three scales are validated and were used preintervention and postintervention. 2.6 | Statistics The data were tested for normality. Data are presented as mean 61 standard deviation (SD) or median with range (min-max). Differences in means, ranks, and ratios were tested by Student’s t test, Mann- Whitney U test, or chi-square test as appropriate. Paired samples t test and Wilcoxon’s signed ranks test were applied for within group comparisons. The null-hypothesis was rejected for P values<.05. All calculations were performed using SPSS 22 (IBM, Armonk, NY, USA). 3 | RESULTS Twenty-three patients were analyzed after follow-up that included 13 in the intervention group and 10 in the control group. There were no differences in baseline data between the intervention group and con- trol group (Tables 1–3). In the population, the mean predicted peak heart rate was 88%67.5% and the mean predicted peak VO 2 was 72%613.7%. Moreover, there were no differences between interven- tion group and control group regarding these data. 3.1 | Exercise capacity The median test duration at constant work rate CPET increased 89% post intervention in the intervention group compared to no change in the control group (median[range] 12[–4 to 52] min vs 0[–4 to 5] min, P5.001). Furthermore, the peak workload (20[–10 to 70] W vs 0[–20 to 15] W, P5.003) at incremental CPET increased post intervention in the intervention group compared to the controls. The peak VO 2 (ml/ min) at incremental CPET increased 15% within the intervention group (P5.019) compared to no change (2%) within the control group (P5.8). The results were similar regarding VO 2 indexed for body 256 | SANDBERG ET AL . weight and peak O 2 pulse. However, in comparison between the inter- vention group and control group no differences were found regarding absolute peak VO 2 (ml/min) or peak VO 2 indexed to body weight (ml/ kg/min) (285[–200 to 535] ml/min vs 17[–380 to 306] ml/min, P5.10) (3.6[–2.6 to 6.4] ml/kg/min vs 0.6[–3.5 to 4.9] ml/kg/min, P5.12). Furthermore, the increase in peak O 2 pulse did not differ between the groups (1.3[–1.7 to 4.2] ml/heartbeat vs 0.4[–1.2 to 2.4], P5.21 (Table 2, Figure 1A–D). 3.2 | EQ-VAS, HADS, and ESE No differences were found within or between groups preintervention and postintervention regarding self-reported QoL, prevalence of anxi- ety and depression or exercise self-efficacy (Table 3). 3.3 | Compliance Compliance to exercise training protocol, defined as the number of completed training sessions in relation to the possible number of ses- sions, was in mean 79%617 (median 83%, 47%–100%). The number of registered exercise occasions ranged from 17 to 36 of 36 possible occasions. 3.4 | Adverse event In one case, the exercise training was discontinued due to the patient experiencing discomfort and possible arrhythmia during a session of exercise training. No arrhythmia was detected on a subsequent exer- cise test or at Holter registration. No other adverse events occurred. TABLE 1 Descriptive data on included patients All patients (n523) Intervention group (n513) Controls (n510) P value Sex M n (%) 12(52) 8(62) 4(40) .31 F n (%) 11(48) 5(39) 6(60) Age, years Median(IQR) 30.1(22.9–36.6) 31.3(26.9–36.6) 26.3(22.9–35.6) .38 Height, m Mean6SD 1.72(0.10) 1.72(0.09) 1.72(0.10) .85 Weight, kg Mean6SD 77(15) 77(10) 76(21) .90 BMI, kg/m 2 Mean6SD 25.8(3.9) 26.0(3.6) 25.5(4) .77 Diagnosis n (%) ToF 5(22) 4(31) 1(10) .19 ccTGA 3(13) 3(23) 0(0) d-TGA 5(22) 2(15) 3(30) TCPC 5(22) 3(23) 2(20) PA 2(9) 0(0) 2(20) Complete AV-septal defect 1(4) 0(0) 1(10) Ebstein 1(4) 0(0) 1(10) Miscellaneous 1(4) 1(8) 0(0) Surgical intervention, yes n (%) 21(91) 11(85) 10(100) .19 Age at intervention, years* median(IQR) 3.1(1.1–6.8) 3.6(1.2–7.6) 3.0(0.7-6.1) .74 PM, yes n (%) 2(9) 2(15) 0(0) .19 Cardiovascular medication, yes n (%) 10(44) 5(39) 5 (50) .58 ACE/ARB n (%) 6(26) 4(31) 2(20) .56 Beta-blockers n (%) 2(9) 1(8) 1(10) .85 Diuretics n (%) 2(9) 1(8) 1(10) .85 Warfarin n (%) 5(22) 3(23) 2(20) .86 Aspirin n (%) 3(13) 2(15) 1(10) .70 Abbreviations: ACE, angiotensin converting enzyme; ARB, angiotensin receptor-2 blockers; AV, atrioventricular; ccTGA, congenitally corrected transposi- tion of the great arteries; d-TGA, dextro-transposition of the great arteries; F, female; IQR, interquartile range; M, male; n, number; PA, pulmonary atre- sia; PM, pacemaker; TCPC, total cavo-pulmonary connection; ToF, tetralogy of Fallot. *Age at TCPC surgery or correction or ToF. Presented P values represent comparison between intervention group and controls. Mann-Whitney U test was applied in comparison of age and age at intervention; in all other comparisons chi-square or Student’s t test was used. SANDBERG ET AL . | 257 4 | DISCUSSION This is the first study to evaluate endurance capacity in addition to peak aerobic capacity after exercise training in adults with complex CHD. The present study shows that home-based high intensity interval training on a cycle ergometer has a great impact on endurance capacity as well as on maximum exercise capacity in adults with complex CHD. 4.1 | Exercise capacity Peak VO 2 (ml/min) increased within the intervention group but not in comparison to the control group. However, the peak work rate for the intervention group increased in comparison to the control group which altogether indicates an improvement in peak aerobic capacity. The increase in peak VO 2 in previous studies was approximately 8%, 14,15,18 and in the present study the corresponding increase within the inter- vention group was 15%. An increase in aerobic capacity could be of significance in daily activities. In patients with complex congenital heart lesions, especially with impaired NYHA class, activities of daily living might be in line with or even exceed their individual exercise capacity. 5 In these cases, an exercise training induced improvement of exercise capacity could play an important role in coping with activities of daily living. In adults with complex CHD, the central adaption to exercise training is usually blunted, 36,37 which might lead peripheral mecha- nisms, that is, increased muscle capillarization and oxidative capacity, to play an even greater role in the response to exercise training. 38 The important increase in endurance capacity may actually reflect this mechanism. It is noteworthy that modest increases in peak VO 2 (ml/ min) and peak work rate after exercise training corresponded to a sub- stantial increase in time duration at constant work rate CPET; this result was previously reported in patients with COPD. 28,39 Our results imply that endurance capacity might be a more clinically relevant mea- sure of change in exercise capacity after exercise training in adults with complex CHD. 4.2 | Exercise testing Peak VO 2 derived from incremental CPET is frequently used and often considered the “gold standard” measure of peak aerobic exercise capacity. 25,26 When assessing the peak aerobic exercise capacity, it is important that the test is performed with maximum effort. The RER?1.10 is considered as a measure of maximum effort being reached. 25 In the present study, this limit was reached by the majority of the participants (Table 2). However, in adults with complex CHD dif- ficulties in reaching this limit was previously reported. 6,40 This phenom- enon was to some extent also observed in our population. Pulmonary limitation of the exercise capacity has been proposed to cause this lim- ited ability to reach RER ?1.10. 25 Recently, submaximal outcome measures calculated from the incremental CPET, that is, ventilatory anaerobic threshold (VAT), oxygen uptake efficiency slope (OUES), and VE/VCO 2 slope, have emerged as useful in evaluation of exercise capacity and as prognostic tools. Furthermore, these parameters do not require a test performed with maximum effort. 26,40,41 In our study, we TABLE 2 Preintervention, postintervention, and change in cardiopulmonary exercise test data in intervention group vs control group Preintervention Postintervention Change after intervention Intervention group Control group P Intervention group Control group P Intervention group Control group P CPET incremental VO 2 peak, ml/min 1865 (1191–2355) 1601 (1215–2650) .74 1870 (1040–2642) 1688 (1326–2367) .26 285 (–200 to 535) 17 (–380 to 306) .10 VO 2 peak, ml/kg/min 23.4 (14.8–29.4) 23.6 (18.1–28.1) .98 26.9 (13.0–33.6) 24.8 (18.1–28.4) .23 3.6 (–2.6 to 6.4) 0.6 (–3.5 to 4.9) .12 Peak O 2 pulse, ml/heartbeat 10.3 (7.3–13.7) 9.5 (6.8–15.1) .99 12.0 (8.3–15.0) 10.8 (8.2–14.0) .48 1.3 (–1.7 to 8.5) 0.4 (–1.2 to 2.4) .21 Peak workload, W 155 (100–220) 150 (110–200) .69 170 (90–240) 140 (110–200) .07 20 (–10 to 70) 0 (–20 to 15) .003 RER 1.19 (0.99-1.51) 1.22 (1.02-1.36) .61 1.20 (1.13-1.40) 1.20 (0.98-1.29) .52 Constant work rate at 75% of peak workload: Test duration min 14 (4–33) 9 (3–20) .11 28 (8–68) 9 (5–16) .001 12 (–4 to 52) 0 (–4 to 5) .001 Abbreviations: CPET, cardiopulmonary exercise test; RER, respiratory exchange ratio. Data are presented as median (range). Bold text indicates a P value<.05. 258 | SANDBERG ET AL . took this a step further and used a submaximal exercise test in addition to the incremental CPET and found a substantially larger (median change 12 min, 89%) increase in submaximal exercise capacity in com- parison to peak VO 2 (15%). With reference to the patients’ perform- ance capacity in daily activities, increased endurance might better illustrate the benefits of improved aerobic capacity and thereby be a more clinically relevant measure. The increase in endurance capacity we found is in line with previous studies in patients with COPD. Pors- zasz et al. 39 reported a mean increase of 11.668.1 minutes in duration at constant work rate CPET after exercise training, while Cambach et al. 42 reported a mean increase of approximately 7 minutes in dura- tion. An increase of 1.6–3.3 minutes has been suggested as a minimal clinically important difference in response to exercise training in patients with COPD. 43,44 In our intervention group, all patients except one increased the time duration at constant work rate CPET above this suggested level of minimal clinical importance. This particular patient did not comply fully with the exercise training protocol fulfilling only 17 of 36 (47%) of the possible exercise training sessions. 4.3 | Exercise training protocol As stated in the current recommendations on physical activity and rec- reational sports in adults with CHD, exercise prescriptions should be individualized. 11 We aimed to provide the patients in the intervention group with an individually adjusted exercise training protocol based on the results of the cardiopulmonary exercise tests. Different modes of exercise training, for example, walking, interval training with step aero- bics, and moderate continuous training on a cycle ergometer have been used in previous studies. 17,18,23,24 The present study is the first to use home-based moderate to high intensity interval exercise training on a cycle ergometer in a population of adults with different complex con- genital heart lesions. Studies in adults with systemic right ventricle have shown that home-based exercise training is safe, feasible and effective with regard to exercise capacity without negative effects on the systemic right ventricle. 17,18,24 In patients with heart failure, inter- val training was reported to improve exercise capacity more than a moderate continuous exercise training mode. 22 The present study FIGURE 1 A-D, Individual cardiopulmonary exercise test data at baseline, at 12-week follow-up and change from baseline to follow-up in intervention group TABLE 3 Preintervention, postintervention and change in data regarding self-reported prevalence of anxiety and depression (HADS), quality of life (EQ5D VAS), and exercise self-efficacy (ESE) Preintervention Postintervention Change after intervention Intervention group Control group P Intervention group Control group P Intervention group Control group P HADS Anxiety 4(1–9) 4(0–7) .69 5(2–9) 6(0–8) .72 1(–3 to 3) 1(0–3) .28 HADS Depression 2(0–9) 2(0–5) .26 2(0–5) 2(0–6) .97 0(–4 to 2) 0(–1 to 4) .18 EQ-VAS 77.5(35.0–99.0) 89.5(70.0–99.0) .10 78.8(48.0–99.0) 85.5(35.0–99.0) .31 0(–21.0 to 25.0) 0(–55.0 to 9.0) .42 ESE 32(16–39) 30(15–40) .88 28(11–40) 29(18–40) .67 25(–12 to 8) 21(–6 to 6) .23 Abbreviations: EQ-VAS, EuroQol Vertical Visual Analogue Scale; ESE, Exercise Self-Efficacy Scale; HADS, Hospital and Anxiety and Depression scale. Data are presented as median (range). SANDBERG ET AL . | 259 showed that home-based interval exercise training increased endur- ance capacity as well as peak aerobic exercise capacity in adults with complex congenital heart lesions. The finding of markedly increased endurance capacity, in addition to peak capacity, contributes important information to previous exercise training studies in this population. 4.4 | Quality of life In CHD, self-reported quality of life is known to be associated with physical activity level. 45 Here, we report that self-reported quality of life remained unchanged after intervention which is in line with results previously shown by others. 15 In contrast, positive effects of exercise on quality of life has been reported. 23 The population in the present study rated their quality of life as rather high preintervention which might explain the unchanged results. 4.5 | Compliance As many patients with complex heart lesions are in the “middle of life” and occupied with studies, work, children, and family activities, a home-based exercise training protocol was chosen to improve adher- ence to study protocol. In addition, participants were contacted by phone every week, which probably also contributed to compliance. The mean compliance in the present study (79%) was in line with previously presented results (68%-77%). 14,17,18 4.6 | Limitations As in previously presented studies on exercise training in adults with CHD, our study population was rather small which somewhat limits the generalizability of the results. However, the results are in line with pre- vious studies which further strengthen exercise training as a part of the rehabilitation of adults with complex CHD. Furthermore, our study population consisted of patients with different complex diagnoses that to a greater extent reflect the diversity seen in the clinic, thus enhanc- ing the generalizability. This study was performed at two centers. The center recruiting 5 patients (22%) was not able to keep the investigators performing the exercise tests strictly blinded for group allocation. However, there was a prespecified protocol that was strictly followed in both centers. Fur- thermore, the same center had a different recruitment strategy that possibly could involve patients with more frequent clinical visits and thus potentially patients with more complex lesions. However, the numbers are small and the two patients performing the exercise proto- col did not obviously differ from the rest of the study population. One concern of the constant work rate CPET that has been dis- cussed previously in patients with COPD is the power/duration rela- tionship. 28 This means that the endurance time varies depending on the workload (power) used during the test. However, there is no con- sensus on optimal power to use which complicates comparison of results between studies. To standardize, we used 75% of peak work rate that was used previously in COPD patients. 39,42 4.7 | Conclusions Home-based interval exercise training increased the endurance capacity at 75% of peak work load by 12 minutes as well as peak exer- cise capacity in adults with different complex congenital heart lesions. Substantially increased endurance capacity in the spectrum of daily activities is what most patients need. Therefore, endurance capacity might be a more clinically relevant target than solely peak oxygen uptake in patients with complex congenital heart lesions. ACKNOWLEDGMENT We are grateful to the personnel at the department of Clinical Phys- iology at the Heart Center, Umeå University Hospital who con- ducted the exercise tests and to the staff at the GUCH-center in G€ oteborg. CLINICAL TRIAL REGISTRATION ClinicalTrials.gov, identification:NCT01671566 CONFLICT OF INTEREST None AUTHOR CONTRIBUTIONS All authors read and approved the final version of the manuscript. Concept/design: Camilla Sandberg, Magnus Hedstr€ om, Karin Wadell, Mikael Dellborg, Bengt Johansson Data collection: Camilla Sandberg, Magnus Hedstr€ om, Mikael Dell- borg, Anders Ahnfelt, Anna-Klara Zetterstr€ om Amanda € Ohrn, Bengt Johansson Data analysis: Camilla Sandberg, Magnus Hedstr€ om, Mikael Dellborg, Bengt Johansson Interpretation: Camilla Sandberg, Magnus Hedstr€ om, Mikael Dellborg, Anders Ahnfelt, Bengt Johansson Drafting: Camilla Sandberg, Karin Wadell, Bengt Johansson Critical revision: Camilla Sandberg, Magnus Hedstr€ om, Karin Wadell, Mikael Dellborg, Anders Ahnfelt, Anna-Klara Zetterstr€ om, Amanda € Ohrn, Bengt Johansson Approval of article: Camilla Sandberg, Magnus Hedstr€ om, Karin Wadell, Mikael Dellborg, Anders Ahnfelt, Anna-Klara Zetterstr€ om, Amanda € Ohrn, Bengt Johansson ORCID Camilla Sandberg RPT, PhD http://orcid.org/0000-0002-4043-7130 REFERENCES [1] Erikssen G, Liestøl K, Seem E, et al. Achievements in congenital heart defect surgery: a prospective, 40-year study of 7038 patients. Circulation. 2015;131(4):337–346. [2] Raissadati A, Nieminen H, Jokinen E, Sairanen H. Progress in late results among pediatric cardiac surgery patients: a population-based 6-decade study with 98% follow-up. Circulation. 2015;131(4): 347–353. 260 | SANDBERG ET AL . [3] Moons P, Bovijn L, Budts W, Belmans A, Gewillig M. Temporal trends in survival to adulthood among patients born with congenital heart disease from 1970 to 1992 in Belgium. Circulation. 2010;122 (22):2264–2272. [4] Fredriksen PM, Veldtman G, Hechter S, et al. Aerobic capacity in adults with various congenital heart diseases. Am J Cardiol. 2001;87 (3):310–314. [5] Kempny A, Dimopoulos K, Uebing A, et al. Reference values for exercise limitations among adults with congenital heart disease. Relation to activities of daily life–single centre experience and review of published data. Eur Heart J. 2012;33(11):1386–1396. [6] Diller GP, Dimopoulos K, Okonko D, et al. Exercise intolerance in adult congenital heart disease: comparative severity, correlates, and prognostic implication. Circulation. 2005;112(6):828–835. [7] Greutmann M, Le TL, Tobler D, et al. Generalised muscle weakness in young adults with congenital heart disease. Heart. 2011;97(14): 1164–1168. [8] Kr€ o€ onstr€ om LA, Johansson L, Zetterstr€ om AK, Dellborg M, Eriksson P, Cider Å. Muscle function in adults with congenital heart disease. Int J Cardiol. 2014;170(3):358–363. [9] Sandberg C, Thil? en U, Wadell K, Johansson B. Adults with complex congenital heart disease have impaired skeletal muscle function and reduced confidence in performing exercise training. Eur J Prev Car- diol. 2014;22(12):1523–1530. [10] Cordina R, O’meagher S, Gould H, et al. Skeletal muscle abnormal- ities and exercise capacity in adults with a Fontan circulation. Heart. 2013;99(20):1530–1534. [11] Budts W, B€ orjesson M, Chessa M, et al. Physical activity in adoles- cents and adults with congenital heart defects: individualized exer- cise prescription. Eur Heart J. 2013;34(47):3669–3674. [12] Duppen N, Takken T, Hopman MTE, et al. Systematic review of the effects of physical exercise training programmes in children and young adults with congenital heart disease. Int J Cardiol. 2013;168 (3):1779–1787. [13] Chaix MA, Marcotte F, Dore A, et al. Risks and benefits of exercise training in adults with congenital heart disease. Can J Cardiol. 2016; 32(4):459–466. [14] Therrien J, Fredriksen P, Walker M, Granton J, Reid GJ, Webb G. A pilot study of exercise training in adult patients with repaired tetral- ogy of Fallot. Can J Cardiol. 2003;19:685–689. [15] Winter MM, van der Bom T, de Vries LC, et al. Exercise training improves exercise capacity in adult patients with a systemic right ven- tricle: a randomized clinical trial. Eur Heart J. 2012;33(11):1378–1385. [16] Cordina RL, O’meagher S, Karmali A, et al. Resistance training improves cardiac output, exercise capacity and tolerance to positive airway pressure in Fontan physiology. Int J Cardiol. 2013;168(2): 780–788. [17] Westhoff-Bleck M, Schieffer B, Tegtbur U, et al. Aerobic training in adults after atrial switch procedure for transposition of the great arteries improves exercise capacity without impairing systemic right ventricular function. Int J Cardiol. 2013;170(1):24–29. [18] Shafer KM, Janssen L, Carrick-Ranson G, et al. Cardiovascular response to exercise training in the systemic right ventricle of adults with transposition of the great arteries. J Physiol. 2015;593 (11):2447–2458. [19] Duppen N, Etnel JR, Spaans L, et al. Does exercise training improve cardiopulmonary fitness and daily physical activity in children and young adults with corrected tetralogy of Fallot or Fontan circula- tion? A randomized controlled trial. Am Heart J. 2015;170(3): 606–614. [20] Duppen N, Geerdink LM, Kuipers IM, et al. Regional ventricular per- formance and exercise training in children and young adults after repair of tetralogy of Fallot: randomized controlled pilot study. Cir- culation Cardiovasc Imaging. 2015;8(4):e002006. [21] Duppen N, Kapusta L, de Rijke YB, et al. The effect of exercise training on cardiac remodelling in children and young adults with corrected tetralogy of Fallot or Fontan circulation: A randomized controlled trial. Int J Cardiol. 2015;179:97–104. [22] Wisløff U, Støylen A, Loennechen JP, et al. Superior cardiovascular effect of aerobic interval training versus moderate continuous train- ing in heart failure patients: a randomized study. Circulation. 2007; 115(24):3086–3094. [23] Dua JS, Cooper AR, Fox KR, Graham Stuart A. Exercise training in adults with congenital heart disease: Feasibility and benefits. Int J Cardiol. 2010;138(2):196–205. [24] Winter MM, van der Bom T, de Vries LC, et al. Exercise training improves exercise capacity in adult patients with a systemic right ventricle: a randomized clinical trial. Eur Heart J. 2011;33(11): 1378–1385. [25] Balady GJ, Arena R, Sietsema K, et al. Clinician’s Guide to cardiopul- monary exercise testing in adults: a scientific statement from the American Heart Association. Circulation. 2010;122(2):191–225. [26] Mezzani A, Agostoni P, Cohen-Solal A, et al. Standards for the use of cardiopulmonary exercise testing for the functional evaluation of cardiac patients: a report from the Exercise Physiology Section of the European Association for Cardiovascular Prevention and Reha- bilitation. Eur J Cardiovasc Prev Rehabil. 2009;16(3):249–267. [27] Clinical exercise testing with reference to lung diseases: indications, standardization and interpretation strategies. ERS Task Force on Standardization of Clinical Exercise Testing. European Respiratory Society. Eur Respir J. 1997;10:2662–2689. [28] Borel B, Provencher S, Saey D, Maltais F. Responsiveness of various exercise-testing protocols to therapeutic interventions in COPD. Pulm Med. 2013;2013:410748. [29] Borg G. Borgs Perceived Exertion and Pain Scales. West Yorkshire, UK: Human Kinetics; 1998:46–49. [30] Ferrazza AM, Martolini D, Valli G, Palange P. Cardiopulmonary exer- cise testing in the functional and prognostic evaluation of patients with pulmonary diseases. Respiration. 2009;77(1):3–17. [31] Kenney WL, Wilmore JH, Costill DL. Physiology of Sport and Exer- cise. 5th ed. West Yorkshire, UK: Human Kinetics; 2012:471–521. [32] The EuroQol Group. EuroQol–a new facility for the measurement of health-related quality of life. Health Policy. 1990;16:199–208. [33] Zigmond AS, Snaith RP. The hospital anxiety and depression scale. Acta Psychiatr Scand. 1983;67(6):361–370. [34] Bjelland I, Dahl AA, Haug TT, Neckelmann D. The validity of the hospital anxiety and depression scale. An updated literature review. J Psychosom Res. 2002;52(2):69–77. [35] Ahlstr€ om I, Hellstr€ om K, Emtner M, Anens E. Reliability of the Swedish version of the Exercise Self-Efficacy Scale (S-ESES): a test- retest study in adults with neurological disease. Physiother Theory Pract. 2015;31(3):194–199. [36] Ohuchi H, Hiraumi Y, Tasato H, et al. Comparison of the right and left ventricle as a systemic ventricle during exercise in patients with congenital heart disease. Am Heart J. 1999;137(6):1185–1194. [37] Norozi K, Wessel A, Alpers V, et al. Chronotropic incompetence in adolescents and adults with congenital heart disease after cardiac surgery. J Card Fail. 2007;13(4):263–268. [38] Bassareo PP, Saba L, Solla P, Barbanti C, Marras AR, Mercuro G. Factors influencing adaptation and performance at physical exercise SANDBERG ET AL . | 261 in complex congenital heart diseases after surgical repair. Biomed Res Int. 2014;2014:862372. [39] Porszasz J, Emtner M, Goto S, Somfay A, Whipp BJ, Casaburi R. Exercise training decreases ventilatory requirements and exercise- induced hyperinflation at submaximal intensities in patients with COPD. Chest. 2005;128(4):2025–2034. [40] Buys R, Cornelissen V, Van De Bruaene A, et al. Measures of exer- cise capacity in adults with congenital heart disease. Int J Cardiol. 2011;153(1):26–30. [41] Reybrouck T, Mertens L, Brusselle S, et al. Oxygen uptake versus exercise intensity: a new concept in assessing cardiovascular exer- cise function in patients with congenital heart disease. Heart. 2000; 84(1):46–52. [42] Cambach W, Chadwick-Straver R, Wagenaar R, van Keimpema A, Kemper H. The effects of a community-based pulmonary rehabilita- tion programme on exercise tolerance and quality of life: a random- ized controlled trial. Eur Respir J. 1997;10(1):104–113. [43] Casaburi R. Factors determining constant work rate exercise toler- ance in COPD and their role in dictating the minimal clinically important difference in response to interventions. COPD. 2005;2(1): 131–136. [44] Laviolette L, Bourbeau J, Bernard S, et al. Assessing the impact of pulmonary rehabilitation on functional status in COPD. Thorax. 2008;63(2):115–121. [45] Sandberg C, Engstr€ om KG, Dellborg M, Thil? en U, Wadell K, Johans- son B. The level of physical exercise is associated with self-reported health status (EQ-5D) in adults with congenital heart disease. Eur J Prev Cardiol. 2015;22(2):240–248. SUPPORTING INFORMATION Additional Supporting Information may be found online in the sup- porting information tab for this article. FIGURE S1 Example of the interval exercise training protocol. The exercise training session had an initial 8 min warm-up without load or with very low load. During the first two weeks, the protocol con- sisted of three intervals and thereafter four intervals. The work load during each interval was adjusted by the patient to reach the indi- vidual training heart rate. The patients were also instructed to reach a perceived exertion corresponding to 14-16 on the Borg scale. The intervals were separated by an active recovery periods of 3 minutes without load or with very low load. Each session ended with a cool down period of approximately 5 min How to cite this article: Sandberg C, Hedstr€ om M, Wadell K, et al. Home-based interval training increases endurance capac`; // Reset `lastIndex` if this regex is defined globally // regex.lastIndex = 0; let m; while ((m = regex.exec(str)) !== null) { // This is necessary to avoid infinite loops with zero-width matches if (m.index === regex.lastIndex) { regex.lastIndex++; } // The result can be accessed through the `m`-variable. m.forEach((match, groupIndex) => { console.log(`Found match, group ${groupIndex}: ${match}`); }); }

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